Is hypertensive vascular disease reversible?
نویسندگان
چکیده
منابع مشابه
Resistance to mineralocorticoid-induced hypertensive vascular disease.
To support our contention that the Wistar-Furth rat is resistant to mineralocorticoid hypertension, we assessed the effects of deoxycorticosterone (DOC) administration or renal artery stenosis on the development of hypertension in the Sprague-Dawley and Wistar-Furth rat strains. Weekly administration of mineralocorticoid in the form of DOC pivalate resulted in rapid, severe hypertensive cardiov...
متن کاملThe Pathology of Hypertensive Pulmonary Vascular Disease
PROGRESSIVE histologic changes occur in the pulmonary arteries and arterioles, as a complication of chronically elevated pulmonary arterial blood pressure, in patients with congenital septal defects of the heart. The progression is so stereotyped as to allow a division of the structural effects into 6 grades (table 1), although it is recognized that there is a gradual change from one grade to a...
متن کاملCLINICOPATHOLOGIC CORRELATIONS Hypertensive Pulmonary Vascular Disease
In an infant with patent ductus arteriosus and pulmonary hypertension, elevated pulmonary vascular resistance persisted following ligation of the ductus. Histologic examination of a pulmonary biopsy at two years of age and tissue obtained at autopsy at three years of age showed obstructive pulmonary vascular disease. The question as to whether the organic pulmonary vascular lesions are secondar...
متن کاملHypertensive Vascular Disease Induced by Heterologous Renin
• Crude rat renin administered to uninephrectomized rats caused hypertensive vascular disease, while semipurified hog renin in equipressor doses, as determined by bio-assay, was ineffective. To explain this difference, the following three possibilities were considered. (1) The lack of activity of hog renin in rats is due to its heterologous nature. (2) Crude rat renin contains an active princip...
متن کاملReversible hypertensive brainstem oedema.
A 60-year-old man presented with a 2-week history of generalised fatigue and bilateral lower limb weakness. He had been found to have hypertension during a health screen performed 1 year previously but was not taking any medications. A physical examination revealed a grade-III hypertensive retinopathy. He had full muscle strength and normal reflexes. His orientation and attention levels were sa...
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ژورنال
عنوان ژورنال: British Journal of Clinical Pharmacology
سال: 1982
ISSN: 0306-5251
DOI: 10.1111/j.1365-2125.1982.tb01346.x